N-Glycan-calnexin interactions in human factor VII secretion and deficiency
نویسندگان
چکیده
منابع مشابه
Jaundice and factor VII deficiency in newborn
Resistance to thyroid hormone is an uncommon condition. We report the clinical and laboratory findings of a case with both resistance to thyroid hormone and, a reduced capacity to produce and respond to thyroid hormone. RTH is a disorder characterized by elevated circulating thyroid hormones, state of non-suppressed pituitary TSH secretion and refractoriness to hormone action in peripheral tiss...
متن کاملjaundice and factor vii deficiency in newborn
resistance to thyroid hormone is an uncommon condition. we report the clinical and laboratory findings of a case with both resistance to thyroid hormone and, a reduced capacity to produce and respond to thyroid hormone. rth is a disorder characterized by elevated circulating thyroid hormones, state of non-suppressed pituitary tsh secretion and refractoriness to hormone action in peripheral tiss...
متن کاملAnticoagulation Therapy Considerations in Factor VII Deficiency
Factor VII (FVII) deficiency is the most prevalent rare bleeding disorder in the USA and affects approximately 1 out of every 500,000 people. Warfarin inhibits the synthesis of FVII, in addition to other clotting factors. Warfarin is contraindicated in patients with bleeding tendencies or blood dyscrasias; therefore, the literature regarding the use of warfarin in FVII deficiency is very limite...
متن کاملFamilial haemophilia and factor VII deficiency.
This investigation is reported because of the combination of familial haemophilia and factor VII deficiency and the unusual occurrence of a female haemophiliac. Cases of combined deficiencies of different clotting factors are extremely rare and up till now there have been reported combinations of haemo-but not of haemophilia and factor VII deficiency. Female haemophiliacs in the homozygous stat...
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ژورنال
عنوان ژورنال: The International Journal of Biochemistry & Cell Biology
سال: 2019
ISSN: 1357-2725
DOI: 10.1016/j.biocel.2019.05.017